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AmNote on Jews and Inherited Mental Illnesses Schizophrenia can causes distorted and paranoid thinking, hallucinations, the hearing of voices, or fixation on single ideas, such as a belief that one is one is an important historical figure or the target of a worldwide conspiracy. If the symptoms of schizophrenia aren't the very definition of Jewish behavior, then nothing is! The Jews' fixation on money alone qualifies them for the loony binbut their international paranoia about their "right to protect themselves", their victimization mania, and their stifling obsession with the Nazis and the "Holocaust" certainly convicts them of ethnic schizophrenia. Schizophrenia is not an absolute, but rather a continuum that all Jews fall into in which even mild incidents or cases of schizophrenia can account for the arrogant delusions of grandeur Jews experience, and their lack of integrity, their lack of respect for others, and their lack of concern about the social consequences of their self-serving behavior to othersantisocial behavior is in the Jews' genes! Schizophrenia is a mechanism that explains the Jew's irrational, inhuman antisocial behavior. Schizophrenia also explains why Jews have a far better opinion of themselves than their deviant behavior warrants, since insanity has robs them of the capacity to make rational judgements, even about themselves. As Dr. Allan Zarkin has proven, Jews are a danger to people, because of their mental illnesses, and so avoid them for your own and your family's safety! All at once Jews are both intelligent and insane, and that is what makes them dangerous. Jews are smart enough to work their way into position of authority, power, and trust, such as doctors, lawyer and, CEOs, yet they don't have the mental capacity of judgement to know right from wrong nor understand the consequences of their actions, since schizophrenia and other mental diseases rob Jews of their mental stability and sense of conscience at any given moment. Such a degenerate mental condition also makes Jews sexually perverted, since they lack the control over good judgement that is necessary for sexual restraint. Religious controlled reproductive behavior based on incest and inbreeding has done the Jews irreparable genetic harm, that has negatively effected their mental stability. The genetic damage has created abnormal brain structures and chemistry, which has resulted in the Jews' inability to interact normally with the rest of humanity. The Jews' abnormal behavior is reflected in their propensity to be political outcasts from the world community and misanthropic behavior that has lead Jews to be viewed as international pariahs. Perhaps, the American military aid used to develop Israeli bio-weapons for killing Blacks and Arabs might be better spent on researching genetic cures for the Jews' numerous mental illnesses, including manic depression. Look into the face of a Jew and you can see the insanity that lurks within himinsanity is in the Jews' blood. Mental illness is the common thread that run through the Jews and binds them together into a pattern of socially destructive behavior. When people refer to Jews as a disease that must be stopped, it turns out that they aren't just speaking figuratively but literally! Jews are a race of mentally ill people bound together and locked into a vicious circle in which behavorial genetics is reenforced by a culture that encourages antisocial and criminal behavior towards others. But to Jews, their aberrant behavior is normal and acceptable, and that is what makes them a risk, since they have some 300 nuclear weapons in their arsenal. America created the problem of Israel, and therefore we must do something to remedy the situation before it is too late for humanity! Scientists are close to discovering a gene that significantly increases the risk of a person developing schizophrenia, one of the most common and debilitating mental illnesses. The results of a 15-year study, involving more than a hundred families and a thousand subjects, has provided researchers with the first definitive evidence that a predisposition to schizophrenia resides on a region of human chromosome 13. Dr Ann Pulver, an epidemiologist at Johns Hopkins University in Baltimore, said: "Finding the actual gene for schizophrenia susceptibility will be like finding a particular house in a large city. "But we've found the city. It's a first step, and an exceedingly important one." The study, published in this month's issue of the journal Nature Genetics, is the first research to provide a genetic "address" for a schizophrenia gene with an internationally accepted degree of statistical reliability. It has been known for many years that schizophrenia tends to run in families. [click here to continue] Researcher Ann Pulver thought she was doing American Jews a favor when she recruited them for a genetic study of schizophrenia and manic depression. She felt it could give Jews a head start in knowing their risk for two potentially devastating mental illnesses. But in Boston and elsewhere, many Jews saw this as yet another chance for the public to stigmatize the genetic health of Jews - and now in the murky world of mental illness. Some Jewish leaders, already worried about discrimination against Jewish women who test positively for a breast-cancer gene mutation, reacted anxiously."Why are we being singled out?" said Nancy Kaufman, director of the Jewish Community Relations Council of Greater Boston. "These studies have created an enormous amount of anxiety in the community." Some Jewish leaders complain that these studies trigger horrific reminders of Nazi mass murders based on the notion of Jewish genetic inferiority. Others are quick to emphasize that today's genetic studies are intended to help people, not hurt them, but genetic privacy is a touchy subject for many Jews: Jewish groups led the recent passage of a Massachusetts genetic privacy law. While at her lab at Johns Hopkins University School of Medicine in Baltimore, Pulver heard a woman over the phone scream at her, "How dare you do this to the Jewish people!" "I'm a Jew," replied Pulver, a response that has often quieted her critics. "Please don't speak for me." American Jews are reassessing their role in today's frenzy over genetic research, trying to decide just what the impact will be if they continue to allow their relatively homogeneous genes to be used in laboratory studies. [click here to continue] Schizophrenia is a common disorder characterized by psychotic symptoms; diagnostic criteria have been established. Family, twin and adoption studies suggest that both genetic and environmental factors influence susceptibility (heritability is approximately 71%; ref. 2), however, little is known about the aetiology of schizophrenia. Clinical and family studies suggest aetiological heterogeneity. Previously, we reported that regions on chromosomes 22, 3 and 8 may be associated with susceptibility to schizophrenia, and collaborations provided some support for regions on chromosomes 8 and 22 (refs 9-13). We present here a genome-wide scan for schizophrenia susceptibility loci (SSL) using 452 microsatellite markers on 54 multiplex pedigrees. Non-parametric linkage (NPL) analysis provided significant evidence for an SSL on chromosome 13q32 (NPL score=4.18; P=0.00002), and suggestive evidence for another SSL on chromosome 8p21-22 (NPL=3.64; P=0.0001). Parametric linkage analysis provided additional support for these SSL. Linkage evidence at chromosome 8 is weaker than that at chromosome 13, so it is more probable that chromosome 8 may be a false positive linkage. Additional putative SSL were noted on chromosomes 14q13 (NPL=2.57; P=0.005), 7q11 (NPL=2.50, P=0.007) and 22q11 (NPL=2.42, P=0.009). Verification of suggestive SSL on chromosomes 13q and 8p was attempted in a follow-up sample of 51 multiplex pedigrees. This analysis confirmed the SSL in 13q14-q33 (NPL=2.36, P=0.007) and supported the SSL in 8p22-p21 (NPL=1.95, P=0.023). [click here to continue] To limit the genetic heterogeneity of schizophrenia, this study focused on the widely extended pedigrees of Ashkenazi Jewish schizophrenia probands. The hypothesis posed is that the increased prevalence among the Ashkenazim of the rare lysosomal enzyme disorders, Tay Sachs disease (TDS), caused by low levels of hexosaminidase A, and Gaucher's disease (GD), caused by low levels of glucocerebrosidase, might contribute to the demonstrated increased vulnerability to schizophrenia in this ethnic group. Signs and symptoms characterizing the candidate illnesses were systematically queried by the family history method. Rates and relative risks for symptoms characterizing these disorders and for several nonautosomal illnesses associated with TSD and/or GD (i.e., amyotrophic lateral sclerosis and Hodgkin's disease, leukemia and lymphoma) are significantly elevated in the schizophrenia pedigrees, compared to controls. The conditions with elevated rates and risks have been associated with chromosomal regions 1q21 and 15q23-q24. These areas are suggested as candidate regions for future targeted deoxyribonucleic acid (DNA) research in schizophrenia. [click here to continue] The heterozygote frequency of Gaucher disease (GD) and Tay-Sachs disease (TSD) is distinctly high among Ashkenazi Jews (1:29 for TSD and 1:16 for GD). Two main theories have been suggested to explain this high occurrence: a founder effect with subsequent genetic drift, and a selective advantage of heterozygotes. We compared the frequency of the GD most common mutation (1226A-->G) among carriers of the common TSD mutation (+1277 TATC) with the frequency of this mutation in the general Ashkenazi population. The frequency of GD carriers among 308 TSD heterozygotes was 1:28 which is about half the expected (P = 0.03). These results indicate that carriers of both diseases do not possess additional evolutionary advantage over single mutation carriers. A reasonable interpretation of these findings is that one or both mutations have arisen relatively recently in different regions of Europe and have not yet reached genetic equilibrium. [click here to continue] The authors compared the rates of treated mental illness among ethnically advantaged Ashkenazic Jews and ethnically disadvantaged Sephardic Jews in four areas of Jerusalem defined by socioeconomic status. They found that the rates of illness were higher in areas of lower socioeconomic status, that the Ashkenazim had higher rates of illness than the Sephardim in the areas of lower status, and that there was a comparatively high rate of illness among the Ashkenazim in the very religious Jewish neighborhoods. They interpret these findings on the basis of the theories of social causation and social selection, including genetic transmission. [click here to continue] Like most announcements and unveilings in the field of genetics, the news that a schizophrenia gene has been identified by scientists needs to be treated with some caution. Imagine a headline reading "Caster sugar identified as causing meringues", and you will have a passable model for how much is often left out in the attempt to compress scientific complexity into a newspaper headline. Nevertheless, the identification by German researchers of a gene implicated in inherited schizophrenia is clearly another step towards a fuller understanding of a disease - another step towards the notional goal of cure or prevention. A clear enough step, anyway, to alarm those for whom the notion of genetic control over human biology has a dark shadow and for whom the notion of "cure" is a decidedly ambiguous one. On Radio 4's Today programme yesterday, Gwynneth Hemmings, of the Schizophrenia Association, warned against simplistic notions that we could eradicate schizophrenia by antenatal screening, for instance. And later this month the biochemist David Horrobin, the association's president, will publish The Madness of Adam and Eve: how schizophrenia shaped humanity, a work that argues that the disease is intimately, even inseparably, linked to the development of human culture. The same mutation that triggered the ascent of man, Horrobin suggests, is responsible for the descent of individual men and women into madness. The emergence of this particular twist of our DNA signalled the divergence "between our large-brained, possibly pleasant, but unimaginative ancestors and the restless, creative creatures that we are today". [click here to continue] One of life's nagging little mysteries is the human urge to sign things just because they are there. With letters, paintings and the like it's logical, acceptable and in most cases, necessary. With natural features like roadside embankments of soft stone, or publicly-funded outdoor furniture, it's illogical, unacceptable and in every case, unnecessary. Yet the members of the ''Kilroy was here'' brigade go on their mindless way, carving, burning, spraying and scratching their progress across the world. Thankfully, a new and more sympathetic environmental consciousness suggests that such vandalism is slowly dying out, at least when it comes to tree-trunks and roadsides, even as the graffiti artists of the cities gain strength and, sometimes, acceptance. Members of the younger generation seem more intent on disfiguring themselves with tattoos, body piercing and even firebrands than on leaving their mark upon the landscape. Where many of the early visitors to the Ngarua Caves on Takaka Hill found it necessary to scribble their names on stalactites and stalagmites, to be gradually preserved in transparent limestone, today's tourists are content to look. Not everyone has got over the urge to record their presence, though, and the most recent example is one of the most bizarre. It is the case of 61-year-old New York obstetrician Allan Zarkin, who is being sued for $ 10 million and faces up to 25 years in prison after admitting that he carved his initials onto a woman's lower abdomen. Last September, just after delivering Liana Gedz's healthy baby by caesarean section, Zarkin picked up his scalpel and committed the sort of crime that figures in the nightmares of people who allow doctors to sedate them, but that is seldom heard of in true life. [click here to continue] It is a good thing, if sometimes painful, to question received wisdom, and Peter Novick does that in spades in "The Holocaust in American Life," a sharp and intelligent inquiry into how the Holocaust has come to be, as Novick says, "our central symbol of atrocity." Novick, a University of Chicago professor who was one of the founders of its program in Jewish studies, does his best to undermine the premise upon which Holocaust obsession and its spawn, guilt, are built. The obsession is recent. After the war, Novick says, Jews shunned identification as victims, and there was almost no public reference to the killing of the Jews. It is only lately, when "the status of victim has come to be prized," that states mandate that the Holocaust be taught in schools, for example. For the survivors themselves, the change can be startling. Forty years ago, Novick observes, they were told that to speak of the Holocaust was bad for them; now it's good. "In both cases, others knew what was best," Novick writes. Public discourse on the subject today is full of contradictions and illogic and, on the part of many Jewish leaders and gentile politicians, self-serving distortions of history, Novick says. These are in no sense justified, he argues, to counter the "tiny band of cranks, kooks and misfits" who deny that the Holocaust occurred, whose influence Novick deems negligible. He objects to the strident insistence (which never permits demur) by many, notably Holocaust survivor Elie Wiesel, that the Holocaust was both utterly unique in the annals of human depravity and of universal significance. How can it be both? Novick asks. Why should non-Jewish Americans mourn Hitler's Jewish victims more than Pol Pot's Cambodian ones? Why is there a Holocaust Museum on the Mall in Washington but no comparable memorial for American slavery? [click here to continue] A book reviewer who deliberately distorts the message of the book he is reviewing in order to serve his own agenda does a disservice to his readers. That is precisely what Michael Lerner did in his review of my book, "The Vanishing American Jew." Incredibly, Lerner tells his readers that had I bothered to "bestir" myself from my "Harvard study," I might have "discovered" the problem that "an obsessive focus on past suffering banishes joy in today's world." Had Lerner bothered to read my book, he would have discovered that this is its central thesis. My entire "Tsuris Theory" is precisely that the Jewish identity is too focused on our past victimization. I explicitly state that "the challenge" confronted by my book is "to move the Jewish state of mind beyond the past obsession with victimization, pain and problems and point it in a new, more positive direction, capable of thriving in an open society." Ironically, Lerner uses the exact word I use--obsessive--when he purports to be telling the reader that I did not say precisely what I did say. I also emphasize the "joy" of Judaism, which he says I ignore. Indeed, I criticize some older Jews for insisting on "focusing on the 'oys' rather than the joys of Judaism." [click here to continue] In her otherwise excellent analysis of extremist views in the aftermath of anti-Semitic attacks in France (''Politics of Bigotry''), Flora Lewis at one point confuses the issues by lumping together statements made by Menachem Begin, Pierre Mendes-France and others. Miss Lewis terms Prime Minister Begin's words in reaction to the bombings in Paris - ''Anti-Semitism and its consequences are no longer an internal affair (of other countries) as far as we are concerned, and we assert the right to intervene to protect Jews wherever they are'' - a ''dangerous doctrine'' and finds it ''ironic that the leader of the Jewish state ... should now claim something like the Pope's universal status to represent his religion.'' I fail to see the irony and the danger. [click here to continue] ![]() |